JOHN B. YOUMANS, M.D., F.A.C.P.; CHARLES TRABUE, M.D.; RALPH S. BUVINGER, M.D.; HELEN FRANK, B.A.
In a previous paper1 it was shown that in human subjects, under basal conditions, ergotamine had little or no effect on those motor divisions of the sympathetic nervous system concerned with the regulation of the blood sugar level or the basal metabolic rate. Evidence was given for the belief that the slowing of the pulse was the result of vagus stimulation rather than depression of the sympathetic. It was suggested at that time that the failure of ergotamine to depress certain of these motor functions of the sympathetic nervous system might be explained if the latter were inactive2 in the
JOHN B. YOUMANS, CHARLES TRABUE, RALPH S. BUVINGER, HELEN FRANK. EXPERIMENTAL AND CLINICAL STUDIES OF ERGOTAMINE: V. THE ACTION OF ERGOTAMINE ON THE SYMPATHETIC NERVOUS SYSTEM STIMULATED BY EPINEPHRINE. STUDIES OF THE METABOLIC RATE, PULSE RATE, BLOOD PRESSURE, BLOOD SUGAR AND THE TOTAL LEUKOCYTE COUNT(V. THE ACTION OF ERGOTAMINE ON THE SYMPATHETIC NERVOUS SYSTEM STIMULATED BY EPINEPHRINE. STUDIES OF THE METABOLIC RATE, PULSE RATE, BLOOD PRESSURE, BLOOD SUGAR AND THE TOTAL LEUKOCYTE COUNT*). Ann Intern Med. 1933;7:653–663. doi: 10.7326/0003-4819-7-5-653
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Published: Ann Intern Med. 1933;7(5):653-663.
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