THOMAS FINDLEY JR., M.D.
Although it has long seemed reasonable to attribute diabetes insipidus to hypofunction of the posterior pituitary it has, until recently, been difficult to understand why lesions limited to the hypothalamus can establish the condition and why on the other hand experimental hypophysectomy does not. The modern theory of diabetes insipidus, as expressed by Ranson and his collaborators whose own brilliant researches1-9 have contributed so much to its conception, goes far toward reconciling such inconsistencies as these and provides at least a working hypothesis susceptible of experimental and clinical demonstration. It rests upon the recent fundamental discovery of a bundle of
FINDLEY T. THYROID-PITUITARY RELATIONSHIP IN DIABETES INSIPIDUS1. Ann Intern Med. ;11:701–713. doi: 10.7326/0003-4819-11-5-701
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Published: Ann Intern Med. 1937;11(5):701-713.
Endocrine and Metabolism, Neurology, Pituitary Disorders.
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