JONATHAN C. MEAKINS, M.D., F.A.C.P.
The recognition of hypoglycemia as the causative factor in the production of a fairly definite group of symptoms has commanded considerable attention in recent years. Both from experimental and clinical observations it has been associated with a wide distribution of pathological lesions. These are tabulated below.
I. New-born infants.
B. Diabetic mother.
A. Experimental ablation (uncontrolled insulin action);
B. Deficient anterior secretion (increased insulin action);
C. Pituitary pancreatropic substance;
D. Simmonds' disease;
E. Basophile adenoma;
F. Adiposogenital dystrophy.
A. Experimental ablation;
JONATHAN C. MEAKINS. HYPOGLYCEMIA FOLLOWING ENCEPHALITIS(HYPOGLYCEMIA FOLLOWING ENCEPHALITIS*). Ann Intern Med. 1940;13:1830–1836. doi: 10.7326/0003-4819-13-10-1830
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Published: Ann Intern Med. 1940;13(10):1830-1836.
CNS Infections, Infectious Disease, Neurology.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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