W. RAAB, M.D.
There exist several experimental methods of producing persistent arterial hypertension: (1) section of the cardio-aortic and carotid sinus "moderator nerves"1-3; (2) impairment of the cerebral blood flow through either intracranial injection of kaolin suspension4 or ligation of most of the vessels which supply the brain circulation5; (3) impairment of the renal blood flow6, 7; (4) repeated intramuscular injections of "hypertonin," which is obtained through ultra-filtration of the serum of hypertensive patients,8 and of various hormonal sterols.165
Elevation of blood pressure following section of the moderator nerves, although a phenomenon of outstanding theoretical interest, apparently has no bearing upon
W. RAAB. THE HORMONAL, CENTRAL AND RENAL ORIGIN OF "ESSENTIAL" HYPERTENSION (CEREBRAL AND RENAL ARTERIOSCLEROTIC ISCHEMIA AS CAUSAL FACTORS)(THE HORMONAL, CENTRAL AND RENAL ORIGIN OF "ESSENTIAL" HYPERTENSION (CEREBRAL AND RENAL ARTERIOSCLEROTIC ISCHEMIA AS CAUSAL FACTORS)*). Ann Intern Med. 1941;14:1981–2007. doi: 10.7326/0003-4819-14-11-1981
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Published: Ann Intern Med. 1941;14(11):1981-2007.
Cardiology, Coronary Risk Factors, Hypertension, Nephrology, Neurology.
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