J. M. LUBITZ; D. W. SCOTT JR.
Confusion still exists concerning the pathogenesis and pathologic picture of the disease complex called Banti's syndrome. Two opposing views are held concerning its development. Banti2 and his adherents considered the splenomegaly to be the primary cause, with liver and vascular changes occurring secondarily. The opposing concept, recently given favor, holds the splenic changes to be secondary to some other cause, usually chronic passive congestion within the portal system. An obstruction is usually described either in the liver, the intra-hepatic blood vessels,1 the portal circulation or the splenic vein itself.
Banti,2 in his original descriptions, pointed out that endophlebitis of the
LUBITZ JM, SCOTT DW. BANTI'S SYNDROME WITH MULTIPLE ANEURYSMS AND THROMBOSES OF THE SPLENIC BLOOD VESSELS1. Ann Intern Med. 1947;26:594–599. doi: 10.7326/0003-4819-26-4-594
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Published: Ann Intern Med. 1947;26(4):594-599.
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