A. ZERNE CHAPMAN, M.D.; PAUL S. REEDER, M.D.; LYLE A. BAKER, M.D., F.A.C.P.
There are few satisfactions in medicine greater than that which comes from seeing a severe neutropenia permanently reverted to normal as a result of therapy. The antibiotics have saved the lives of many of these patients during their acute infections, but the ultimate aim is to return the granulocytes to normal in number and activity. The reversible neutropenias are frequently associated with splenomegaly in which splenectomy is often curative. Such a hypersplenic mechanism, associated with a tuberculous etiology, is not common and, when present, is usually found in the disseminated or miliary form of tuberculosis. The case presented is of
A. ZERNE CHAPMAN, PAUL S. REEDER, LYLE A. BAKER. NEUTROPENIA SECONDARY TO TUBERCULOUS SPLENOMEGALY: REPORT OF A CASE(NEUTROPENIA SECONDARY TO TUBERCULOUS SPLENOMEGALY: REPORT OF A CASE*). Ann Intern Med. 1954;41:1225–1231. doi: 10.7326/0003-4819-41-6-1225
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Published: Ann Intern Med. 1954;41(6):1225-1231.
Infectious Disease, Mycobacterial Infections.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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