HENRY BLACKBURN, M.D.; JOSEF BROŽEK, Ph.D.; HENRY L. TAYLOR, Ph.D.
Tobacco smoking has long been suspected as an etiologic factor in respiratory symptoms and chronic bronchopulmonary disease, largely on the basis of clinical impression. Recent evidence tends to confirm this impression. Auerbach and associates reported greater prevalence of pathologic changes in the bronchial mucosa of smokers than in nonsmokers.1 Eich, Gilbert and Auchincloss found increased airway resistance as an acute effect of cigarette smoking in a few patients with pulmonary emphysema. They observed no change in pulmonary mechanics after smoking in normal subjects.2 In Higgins' field study of a random population sample in England there was evidence of reduced ventilatory
HENRY BLACKBURN, JOSEF BROŽEK, HENRY L. TAYLOR. LUNG VOLUME IN SMOKERS AND NONSMOKERS(LUNG VOLUME IN SMOKERS AND NONSMOKERS*†)(LUNG VOLUME IN SMOKERS AND NONSMOKERS*†). Ann Intern Med. 1959;51:68–77. doi: 10.7326/0003-4819-51-1-68
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Published: Ann Intern Med. 1959;51(1):68-77.
Cardiology, Coronary Risk Factors, Smoking, Tobacco, Alcohol, and Other Substance Abuse.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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