CHANYO BENYAJATI, M.D.; MUNI KEOPLUG, M.D.; WILLIAM R. BEISEL; EUGENE J. GANGAROSA; HELMUTH SPRINZ, F.A.C.P.; VISITH SITPRIJA, M.D.
Progressive uremia has long been recognized as a late and often fatal complication of Asiatic cholera. In 1921, anuria in cholera was attributed by Rogers1 to a fall in blood pressure leading to impaired circulation through the kidneys. Two decades later, Tomb2, 3 stated that the renal pathologic changes in cholera resembled those seen in renal anoxia, a concept supported by Wilkinson4 and by Maegraith et al.5 De, Sengupta et al.6 further clarified the pathogenesis by differentiating between the typical initial anuria of the hemoconcentrated cholera patient with circulatory collapse and the less common occurrence of true acute renal failure.
BENYAJATI C, KEOPLUG M, BEISEL WR, GANGAROSA EJ, SPRINZ H, SITPRIJA V. ACUTE RENAL FAILURE IN ASIATIC CHOLERA: CLINICOPATHOLOGIC CORRELATIONS WITH ACUTE TUBULAR NECROSIS AND HYPOKALEMIC NEPHROPATHY(ACUTE RENAL FAILURE IN ASIATIC CHOLERA: CLINICOPATHOLOGIC CORRELATIONS WITH ACUTE TUBULAR NECROSIS AND HYPOKALEMIC NEPHROPATHY*). Ann Intern Med. 1960;52:960–975. doi: 10.7326/0003-4819-52-5-960
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Published: Ann Intern Med. 1960;52(5):960-975.
Infectious Disease, Nephrology.
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