ROBERT C. HINMAN, M.D.; KENNETH R. MAGEE, M.D., F.A.C.P.
Classification of the Guillain-Barré syndrome has been the subject of much discussion and review (1-11). The diversity of the clinical manifestations has led some clinicians to establish criteria that would more precisely delimit the syndrome (1). Others take issue with this restrictive tendency, believing that it excludes certain clinical manifestations such as profound sensory loss, relapses, and ataxia that should be considered part of the syndrome (2-5, 12). This attempt at restrictive classification of the syndrome would be securely based if the etiologic mechanism(s) were understood. Despite the widely accepted parainfectious etiologic component, it is rarely possible to establish a
ROBERT C. HINMAN, KENNETH R. MAGEE. Guillain-Barré Syndrome with Slow Progressive Onset and Persistent Elevation of Spinal Fluid Protein. Ann Intern Med. 1967;67:1007–1012. doi: 10.7326/0003-4819-67-5-1007
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Published: Ann Intern Med. 1967;67(5):1007-1012.
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