This content is PDF only. Please click on the PDF icon to access.
After a brief and historically oriented review of the topic, the authors' view of pathogenesis in paroxysmal supraventricular tachycardia is presented, and it may be summarized as follows. In the majority of cases, hypofunction of the pituitary or of its target glands induces hypothalamic excitation, which not only serves to stimulate endocrine activity but also spreads to cell groups controlling vegetative activity and thus induces tachycardia. This hypothesis receives support, the authors claim, from beneficial results of endocrine substitution therapy including pituitary implantation. To me, this aspect of the book is somewhat less than convincing.
Case histories of 68 patients
Paroxysmal Tachycardia: Pathogenesis and Treatment of the Supraventricular Form.. Ann Intern Med. ;68:1377–1378. doi: 10.7326/0003-4819-68-6-1377_2
Download citation file:
Published: Ann Intern Med. 1968;68(6):1377-1378.
Copyright © 2018 American College of Physicians. All Rights Reserved.
Print ISSN: 0003-4819 | Online ISSN: 1539-3704
Conditions of Use