Gabriel H. Schwartz, M.D.; Robert R. Riggio, M.D.; Paul Saville, M.D.; John Whitsell, M.D.; Kurt H. Stenzel, M.D.; Albert L. Rubin, M.D., F.A.C.P.
This content is PDF only. Please click on the PDF icon to access.
Hypophosphatemia and several types of hypercalcemia after renal transplantation are described. Serum and urine calcium, phosphorus, creatinine, and serum parathormone (PTH) (measured by Dr. Yalow) were followed on 25 transplanted patients. Hypophosphatemia occurred in nearly all patients and correlated with the administration of oral hydroxide antacids. Clinical effects of phosphorus depletion included weakness, intention tremor, bone pain, pseudofractures, and hypercalcemia. Treatment with oral phosphate reversed the abnormalities. Aluminum phosphate gel did not induce hypophosphatemia. Hypercalcemia of 12 to 15 mg/100 ml necessitated subtotal parathyroidectomy in five patients, two of whom had oliguria and one, polyuria due to hypercalcemia. Failure to
Schwartz GH, Riggio RR, Saville P, Whitsell J, Stenzel KH, Rubin AL. Hypercalcemia, Hypophosphatemia, and Renal Transplantation.. Ann Intern Med. 1969;70:1096. doi: 10.7326/0003-4819-70-5-1096_2
Download citation file:
Published: Ann Intern Med. 1969;70(5):1096.
Endocrine and Metabolism, Fluid and Electrolyte Disorders, Nephrology.
Results provided by:
Copyright © 2018 American College of Physicians. All Rights Reserved.
Print ISSN: 0003-4819 | Online ISSN: 1539-3704
Conditions of Use