VICTOR E. POLLAK, M.D., F.A.C.P.
That glomerulonephritis can be produced experimentally by immunologic mechanisms is now well known, and there is much evidence to suggest that similar mechanisms are responsible for some types of glomerular disease in man (1). More recent is the view that coagulation within the glomerular capillaries may play an important role in the pathogenesis of certain types of glomerular changes. This view is based on the demonstration of fibrinogen derivatives and of fibrin and platelet thrombi in the kidney and particularly within the glomerular capillaries (2-5). Increasing evidence supports the occurrence in some renal diseases of products of fibrin breakdown in
POLLAK VE. Anticoagulants in the Treatment of Renal Disease. Ann Intern Med. 1970;73:334–335. doi: 10.7326/0003-4819-73-2-334
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Published: Ann Intern Med. 1970;73(2):334-335.
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