JACOB D. BITRAN, M.D.
To the editor: The recent paper by Koffler, Friedler, and Massry (Ann Intern Med 85:23-28, 1976) attributes hypercalcemia in the diuretic phase of acute renal failure to either secondary hyperparathyroidism, immobilization, or remobilization of previously deposited calcium in damaged muscle.
A patient we have recently seen who presented with acute renal failure from alcoholic rhabdomyolysis had clearly elevated plasma parathyroid hormone levels throughout his course and well into the diuretic phase without the development of hypercalcemia (Table 1). The parathyroid hormone level was inappropriately elevated in relation to the serum calcium or magnesium level and suggests loss of normal feedback
BITRAN JD. Secondary Hyperparathyroidism in Acute Renal Failure from Rhabdomyolysis. Ann Intern Med. ;85:679. doi: 10.7326/0003-4819-85-5-679_1
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Published: Ann Intern Med. 1976;85(5):679.
Acute Kidney Injury, Endocrine and Metabolism, Nephrology, Parathyroid Disorders.
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