DAVID H. P. STREETEN, M.B., D.PHIL.; GUNNAR H. ANDERSON JR., M.D.
For many years it has been known that plasma renin activity was elevated in some forms of human hypertension and that angiotensin II, the end-product of the reactions initiated by renin, could be the cause of such types of hypertension. Because the blood pressure has not always been raised when plasma renin activity was above normal, however, as in Bartter's syndrome or cirrhosis with ascites, it has been difficult to know what role, if any, was played by renin and angiotensin excess in the genesis of hypertension. The recent use of angiotensin blockade with competitive antagonists of angiotensin II, like
STREETEN DHP, ANDERSON GH. Angiotensin Blockade in Hypertension. Ann Intern Med. ;86:353–354. doi: 10.7326/0003-4819-86-3-353
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Published: Ann Intern Med. 1977;86(3):353-354.
Cardiology, Coronary Risk Factors, Hypertension, Infectious Disease, Nephrology.
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