WILLIAM B. RAWLS, M.D., F.A.C.P.; SAMUEL WEISS, M.D., F.A.C.P.; VERA L. COLLINS, M.D.
During the past few years, much interest has been focused on the subject of decreased dextrose tolerance in acute infectious diseases. Williams and Dick1 found a decreased tolerance in 41 per cent of patients and they also found a similar disturbance in carbohydrate metabolism in experimentally induced toxemia in animals. Their paper contains an excellent review of the literature.
Two explanations have been offered for the "diabetic" type of dextrose tolerance curves obtained in these conditions. Some authors1, 2, 3 ascribe it to lack of endogenous insulin caused by the functional impairment of the islets of Langerhans, while others4, 5,
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RAWLS WB, WEISS S, COLLINS VL. LIVER FUNCTION IN RHEUMATOID (CHRONIC INFECTIOUS) ARTHRITIS; PRELIMINARY REPORT(LIVER FUNCTION IN RHEUMATOID (CHRONIC INFECTIOUS) ARTHRITIS; PRELIMINARY REPORT*). Ann Intern Med. 1937;10:1021–1027. doi: 10.7326/0003-4819-10-7-1021
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Published: Ann Intern Med. 1937;10(7):1021-1027.
Rheumatoid Arthritis, Rheumatology.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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