R. W. KISSANE, M.D., F.A.C.P.; R. S. FIDLER, B.A., M.D.; R. A. KOONS, B.A., M.D.
Medical literature contains considerable evidence, obtained by autopsy, that non-penetrating wounds to the chest can produce severe traumatic lesions in the pericardium, heart and great vessels, but it is not generally accepted that less severe trauma can cause mild changes in the heart which have a tendency to recover.
Beck1 described experiments on dogs where the heart was exposed and contused. The resultant myocardial injuries produced electrocardiographic changes similar to those obtained in coronary artery occlusion, affecting chiefly the Q- and T-wave and having a tendency to return to normal. Randles, Gorham and Dresbach2 described alternation in the RS-T component
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KISSANE RW, FIDLER RS, KOONS RA. ELECTROCARDIOGRAPHIC CHANGES FOLLOWING EXTERNAL CHEST INJURY TO DOGS(ELECTROCARDIOGRAPHIC CHANGES FOLLOWING EXTERNAL CHEST INJURY TO DOGS*). Ann Intern Med. 1937;11:907–935. doi: 10.7326/0003-4819-11-6-907
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Published: Ann Intern Med. 1937;11(6):907-935.
Cardiac Diagnosis and Imaging, Cardiology, Emergency Medicine.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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