MATTHEW TAUBENHAUS, M.D., F.A.C.P.; ORMAND C. JULIAN, M.D., F.A.C.S.; JAKUB SCHLICHTER, M.D.; MARGARET LITTMAN, M.D.
Interest in the problem of portal hypertension has been renewed since successful new operative procedures have been devised to relieve the increased pressure in the portal system.1, 2, 3, 4, 5 The recognition of this syndrome presents no major problem, particularly if a stage is reached in which pronounced signs and symptoms are present. These consist of a history of hematemesis and/or melena, the presence of ascites, splenomegaly and enlarged collateral veins in the abdominal wall, and x-ray evidence of esophageal varices. The anatomic basis for this condition has been described6 the experimental work on this subject reviewed,7, 8 and
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TAUBENHAUS M, JULIAN OC, SCHLICHTER J, LITTMAN M. UNUSUAL ETIOLOGIES OF PORTAL SYSTEM HYPERTENSION (TORULOSIS, PHLEBOSCLEROSIS AND RETROPERITONEAL CHRONIC INFLAMMATION)(UNUSUAL ETIOLOGIES OF PORTAL SYSTEM HYPERTENSION (TORULOSIS, PHLEBOSCLEROSIS AND RETROPERITONEAL CHRONIC INFLAMMATION)*). Ann Intern Med. 1954;40:313–326. doi: 10.7326/0003-4819-40-2-313
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Published: Ann Intern Med. 1954;40(2):313-326.
Cardiology, Coronary Risk Factors, Hypertension, Infectious Disease, Nephrology.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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