W. B. YOUMANS, M.D., Ph.D.
From time to time physicians have noted certain similarities between the syndrome produced by a systemic arteriovenous fistula and the cardiovascular effects produced by chronic thiamine deficiency.1 Not only do the circulatory patterns in these two conditions resemble each other, but also the circulatory changes produced by therapy with large doses of thiamine in patients with beriberi heart disease are similar in several respects to those associated with closure of an arteriovenous fistula.1 It has become common practice to refer to the circulatory failure of the type produced by beriberi and arteriovenous fistula as high output failure, in recognition of
Learn more about subscription options.
Register Now for a free account.
YOUMANS WB. MECHANISM OF HIGH OUTPUT CIRCULATORY FAILURE(MECHANISM OF HIGH OUTPUT CIRCULATORY FAILURE*). Ann Intern Med. 1954;41:747–756. doi: 10.7326/0003-4819-41-4-747
Download citation file:
Published: Ann Intern Med. 1954;41(4):747-756.
Results provided by:
Copyright © 2017 American College of Physicians. All Rights Reserved.
Print ISSN: 0003-4819 | Online ISSN: 1539-3704
Conditions of Use
This PDF is available to Subscribers Only