JAMES F. MORRISSEY, M.D.; MANUEL OCHOA JR., M.D.; WILLIAM D. LOTSPEICH, M.D.; CHRISTINE WATERHOUSE, M.D., F.A.C.P.
Metabolic and respiratory acidosis has been shown to decrease the urinary citrate excretion in man (1). Conversely, metabolic or respiratory alkalosis will increase this parameter, without associated increase in the plasma citrate level (2). In these situations, usually the urine pH varies directly with the pH of the blood which does not allow one to differentiate the significance of either factor in the renal excretion of citrate. Recently, Evans, Maclntyre, Macpherson, and Milne (2) have shown that potassium repletion in a patient with primary aldosteronism was associated with an increased urinary citrate excretion. In their case, this occurred in the
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MORRISSEY JF, OCHOA M, LOTSPEICH WD, WATERHOUSE C. Citrate Excretion in Renal Tubular Acidosis. Ann Intern Med. 1963;58:159–166. doi: 10.7326/0003-4819-58-1-159
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Published: Ann Intern Med. 1963;58(1):159-166.
Endocrine and Metabolism, Nephrology.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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