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Despite the efforts of many investigators our ability to alter the course of coronary atherosclerosis remains severely limited. This unhappy situation reflects the enormous complexity of a human disease process that requires decades to evolve, can be examined directly only in its final, fatal stage of evolution, and for which no entirely suitable animal model exists. It seems improbable that this problem, so unlikely of successful challenge, would have attracted the efforts of so many investigators had it not been our nation's primary cause of death.
Viewed in this context, it is understandable that several lines of investigation—animal model, biochemical,
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Pathogenesis of Coronary Artery Disease.. Ann Intern Med. 1970;73:151–152. doi: 10.7326/0003-4819-73-1-151
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Published: Ann Intern Med. 1970;73(1):151-152.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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