PETER A. SCHLESINGER, M.D.; PAUL H. DURAY, M.D.; BARBARA A. BURKE, M.D.; ALLEN C. STEERE, M.D.; M. THOMAS STILLMAN, M.D.
SCHLESINGER PA, DURAY PH, BURKE BA, STEERE AC, STILLMAN MT. Maternal-Fetal Transmission of the Lyme Disease Spirochete, Borrelia burgdorferi. Ann Intern Med. 1985;103:67-68. doi: 10.7326/0003-4819-103-1-67
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Published: Ann Intern Med. 1985;103(1):67-68.
Lyme disease usually begins with a characteristic skin lesion, erythema chronicum migrans, accompanied by "influenza-like" or "meningitis-like" symptoms (1). Some patients later develop cardiac abnormalities such as atrioventricular heart block or myopericarditis, neurologic complications, or intermittent attacks of arthritis (1). The causative agent, the Lyme disease spirochete Borrelia burgdorferi (2), is transmitted by Ixodes dammini or related ixodid ticks (3). Antibiotic treatment with tetracycline or penicillin is usually curative (4).
We report the case of a woman who developed Lyme disease during the first trimester of pregnancy. She did not receive antibiotic therapy. Her infant, born at 35 weeks gestational
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Infectious Disease, Tick-Borne Diseases.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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