Kin-Kee Pun, MD; Cheuk-Kit Wong, MD; Elaine Yee-Ling Tsui, MD; Sidney Chi-Fai Tam, MD; Annie Wai-Chee Kung, MD; Christina Chun-Lun Wang, MD
Pun K, Wong C, Tsui EY, Tam SC, Kung AW, Wang CC. Hypokalemic Periodic Paralysis due to the Sjögren Syndrome in Chinese Patients. Ann Intern Med. 1989;110:405-406. doi: 10.7326/0003-4819-110-5-405
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Published: Ann Intern Med. 1989;110(5):405-406.
The Sjögren syndrome is complicated by distal renal tubular acidosis in about 40% of the patients (1). Three patients presented with hypokalemic paralysis as a result of renal tubular acidosis that occurred 2 to 7 years before xerostomia or xerophthalmia. Hypokalemic paralysis occurs more frequently in Orientals as a result of thyrotoxicosis (2, 3), hyperaldosteronism (4), barium poisoning (5), and as a side effect of glossypol, a fertility regulating agent used in China (6).
A 34-year-old southern Chinese woman was hospitalized because of sudden onset of quadriparesis caused by exertion followed by a heavy meal. On admission, there was
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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