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Hyperparathyroidism: Analysis of Recent Experiences, Clinical Spectrum, and Diagnostic Tests

Ann Intern Med. 1962;56(2):183-197. doi:10.7326/0003-4819-56-2-183
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Since mandl performed the first parathyroidectomy in the treatment of osteitis fibrosa cystica a little more than a quarter of a century ago (1), clinical concepts of primary hyperparathyroidism have changed markedly. The frequency of renal calculi as a common complication of primary parathyroid hyperfunction was recognized early (2), and calculi continue to be the most important single clue arousing suspicion of hyperparathyroidism. In recent years, the frequency of gastrointestinal complications has been emphasized by several observers (3-6). Mental aberrations (7, 8), pancreatitis (9), occasional familial tendency (10-12), hypertension (13), and association with tumors of other endocrine organs (14, 15)


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