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Idiopathic Dyserythropoietic Jaundice.

S. Berendsohn, M.D.; J. Lowman, M.D.; R. D. Sundberg, M.D., Ph.D.; and C. J. Watson, M.D., M.A.C.P.
Ann Intern Med. 1963;58(4):732. doi:10.7326/0003-4819-58-4-732_2
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A 35-year-old Peruvian male with prolonged retention jaundice has been studied in detail with special reference to the origin of the great excess of bilirubin in the serum and bile, and of urobilinogen in the feces. Superficially, the disease simulated hemolytic jaundice although the moderate anemia was normocytic normochromic without increased fragility or spherocytosis of the red blood cells. Thalassemia was excluded.

With use of N15 glycine it was readily established that the life span of the circulating erythrocytes was normal and that nearly all of the fecal urobilinogen was derived from sources other than the hemoglobin of mature circulating




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