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Ankylosing Spondylitis, Hyperuricemia, and Anomalies of the Lumbosacral Junction: A Family Study.

George R. Thompson, M.D.; Milovan T. Rakic, M.D.; William Martel, M.D.; and John T. Hayes, M.D.
Ann Intern Med. 1964;60(2_Part_1):315. doi:10.7326/0003-4819-60-2-315_1
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It is generally accepted that genetic factors are important in ankylosing spondylitis, hyperuricemia, and gout. While it is probably not a unique association, to our knowledge, there are no reports of these illnesses occurring in the same patient. It therefore seems pertinent to call attention to a family in which hyperuricemia and gout, ankylosing spondylitis, and various developmental anomalies of the lower spine occurred in several members of three generations.

The propositus, a 44-year-old man, had tophaceous gout, ankylosing spondylitis, and a transitional lumbosacral vertebra with spine bifida. Of his eight children, two had ankylosing spondylitis, one of whom also


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