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Multiple Endocrine Organ Refractoriness to Trophic Hormone Stimulation: A Patient With an Enlarged Sella Turcica and Increased FSH Secretion

STEPHEN J. GORDON, M.D.; and ARNOLD M. MOSES, M.D.
Ann Intern Med. 1965;63(2):313-316. doi:10.7326/0003-4819-63-2-313
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This excerpt has been provided in the absence of an abstract.

Pituitary tumors are usually nonfunctioning, the associated loss of end organ function being due to diminished trophic hormone secretion. It has been noted, however, that single primary end organ failure can result in production of a trophic hormone producing tumor (1). The following is a report of a patient initially thought to have a pituitary tumor with hypopituitarism but who had increased follicle-stimulating hormone (FSH) excretion and no thyroidal, adrenal, or gonadal responsiveness to trophic hormone stimulation. We believe this is the first such case to be reported.

CASE HISTORY: Patient C. F., a 47-year-old white male, was first admitted

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