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Abstracts |

Hypercalcemia, Hypophosphatemia, and Renal Transplantation.

Gabriel H. Schwartz, M.D.; Robert R. Riggio, M.D.; Paul Saville, M.D.; John Whitsell, M.D.; Kurt H. Stenzel, M.D.; and Albert L. Rubin, M.D., F.A.C.P.
Ann Intern Med. 1969;70(5):1096. doi:10.7326/0003-4819-70-5-1096_2
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Hypophosphatemia and several types of hypercalcemia after renal transplantation are described. Serum and urine calcium, phosphorus, creatinine, and serum parathormone (PTH) (measured by Dr. Yalow) were followed on 25 transplanted patients. Hypophosphatemia occurred in nearly all patients and correlated with the administration of oral hydroxide antacids. Clinical effects of phosphorus depletion included weakness, intention tremor, bone pain, pseudofractures, and hypercalcemia. Treatment with oral phosphate reversed the abnormalities. Aluminum phosphate gel did not induce hypophosphatemia. Hypercalcemia of 12 to 15 mg/100 ml necessitated subtotal parathyroidectomy in five patients, two of whom had oliguria and one, polyuria due to hypercalcemia. Failure to


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