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Hyperparathyroidism and Carcinoid Tumor

NAGUIB A. SAMAAN, M.D., Ph.D., F.A.C.P., F.R.C.P.; ROBERT C. HICKEY, M.D., F.A.C.S.; TOM D. BEDNER, M.D.; and MICHAEL L. IBANEZ, M.D.
[+] Article and Author Information

Grant support: grant CA-05831-13, National Institutes of Health; grant CI-78B, American Cancer Society; and the U.S. Public Health Service.

▸Reprint requests should be addressed to Naguib A. Samaan, M.D., Ph.D., F.A.C.P., Chief, Section of Endocrinology, M. D. Anderson Hospital, 6723 Bertner Ave., Houston, TX 77025.


Houston, Texas


Ann Intern Med. 1975;82(2):205-207. doi:10.7326/0003-4819-82-2-205
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Three patients with carcinoid tumor associated with hyperparathyroidism are described. All patients showed a high circulating immunoreactive calcitonin level with no differential increase in the neck venous catheterization specimens, suggesting that the high concentrations of circulating immunoreactive calcitonin may have come from the carcinoid tumor. We think that the hyperparathyroidism in these patients was primary and that the association with carcinoid tumor represents another form of multiple endocrine tumor formation. Hyperparathyroidism should be investigated in patients with carcinoid tumor.

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