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Plasma Triglyceride and Insulin Levels in Familial Hypertriglyceridemia

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This study was supported in part by NIH grant HL-18687 and AM-02456. Part of this work was done in the Clinical Research Center at the University Hospital (FR-37). The authors wish to thank Mr. Howard Beiter and Ms. Martha Kimura for their assistance.

Division of Metabolism, Endocrinology, and Gerontology
Department of Medicine
University of Washington
Seattle, WA 98195

Ann Intern Med. 1977;87(2):198-199. doi:10.7326/0003-4819-87-2-198
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This excerpt has been provided in the absence of an abstract.

Plasma triglyceride levels have been correlated with insulin levels in both normal populations and in patients with endogenous hypertriglyceridemia (1). As a result of this reproducible significant association, it has been suggested that hyperinsulinism might be causally related to endogenous hypertriglyceridemia (2).

It has now been established that "endogenous hypertriglyceridemia" describes a heterogenous group of primary familial and sporadic disorders. Recently two common forms of monogenic hypertriglyceridemia have been described: "pure" monogenic familial hypertriglyceridemia, in which the hypertriglyceridemic propositus comes from a family in which all affected relatives have isolated hypertriglyceridemia; and familial combined hyperlipidemia, in which the hypertriglyceridemic propositus


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