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Serum Vanadium Levels in Chronic Renal Disease

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Departments of Physiology and Nephrology, Washington University School of Medicine and the Jewish Hospital of St. Louis; St. Louis, Missouri

Ann Intern Med. 1979;91(5):743. doi:10.7326/0003-4819-91-5-743
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Patients with progressive renal failure maintain sodium balance by excreting a higher fraction of the filtered load of sodium (1). Although the mechanisms responsible for the increase in fractional sodium excretion are not completely understood, a number of studies suggest that an inhibitor(s) of sodium transport is present in the serum and urine from patients with chronic renal failure (2, 3). Recently, it has been shown that sodium vanadate, an inhibitor of (Na+, K+)-activated adenosine triphosphatase, has a potent natriuretic effect in the rat (4, 5). Because vanadium is a naturally occurring trace element and is excreted by the kidney,


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