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Hepatitis B Surface Antigen in Urine of Renal Transplant Recipients

LANA KAISER, M.D.; THOMAS J. KELLY, B.S.; MARIA J. PATTERSON, Ph.D.; TOM V. SANCHEZ, B.S.; RONALD S. SHAPIRO, M.D.; and GILBERT H. MAYOR, M.D.
[+] Article and Author Information

Supported by a grant from the Michigan Department of Public Health.

▸Requests for reprints should be addressed to Gilbert H. Mayor, M.D.; B220 Life Sciences, Department of Medicine, Michigan State University; East Lansing, MI 48824.


Michigan State University; East Lansing, Michigan. Medical College of Ohio; Toledo, Ohio


Ann Intern Med. 1981;94(6):783-784. doi:10.7326/0003-4819-94-6-783
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This excerpt has been provided in the absence of an abstract.

Urinary hepatitis B surface antigen (HBsAg) has been shown by radioimmunoassay in 19 of 36 persistent serum HBsAg carriers on maintenance hemodialysis (1); however, data on antigenuria in transplant recipients are less conclusive. Blainey and associates (2) detected low levels of urinary antigen by complement fixation in each of seven transplant recipients without reference to renal function or prior bilateral nephrectomy, therefore, not excluding the in-situ end-stage kidneys as the source of antigenuria. The only previous report of antigenuria measured by radioimmunoassay in a transplant patient with prior bilateral nephrectomy links an adequately functioning allograft to intermittent passage of urinary

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