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Staphylococcus Aureus and Toxic Shock Syndrome |

Possible Host-Defense Mechanisms in Toxic Shock Syndrome

JEFFREY P. DAVIS, M.D.; JAMES M. VERGERONT, M.D.; and P. JOAN CHESNEY, M.D.
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▸Requests for reprints should be addressed to Jeffrey P. Davis, M.D.; Bureau of Community Health and Prevention, Division of Health, P.O. Box 309; Madison, WI 53701.


Madison, Wisconsin


Ann Intern Med. 1982;96(6_Part_2):986-991. doi:10.7326/0003-4819-96-6-986
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Future research activity in toxic shock syndrome should provide further understanding of the ways different host-defense mechanisms are involved in toxic shock syndrome; such efforts must consider toxic shock syndrome as a toxin-mediated disease. Topics that require further research include Staphylococcus aureus acquisition in relation to toxic shock syndrome; bacterial colonization factors including bacterial adherence, vaginal mucosal alteration, secretory IgA, and indigenous flora; conditions that affect organism growth and toxin production including the presence of iron, the availability of calcium, potential defects in opsonization and neutrophil function, and potential defects in lymphocyte function; and the development of natural antibodies against a suspected toxin associated with toxic shock syndrome.

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