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Reactivation of Type 2 Herpes Simplex Virus by Thoracolumbar Neurosurgery

HARRY W. HAVERKOS, M.D.; GEORGE J. PAZIN, M.D.; MONTO HO, M.D.; and PAUL B. NELSON, M.D.
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University of Pittsburgh and Presbyterian-University Hospital; Pittsburgh, Pennsylvania.


Ann Intern Med. 1984;101(4):503-504. doi:10.7326/0003-4819-101-4-503
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Orofacial Herpes Simplex Virus recurrences after neurosurgery on the trigeminal sensory root (1-5) suggested a relationship between recurrent herpes labialis, herpes simplex virus latency, and the trigeminal nerve that was confirmed by detection of latent herpes simplex virus in trigeminal ganglia (6-8). Similarly, latency of type 2 herpes simplex virus in sacral ganglia of humans has been shown by Baringer (9). We report three patients with reactivation of type 2 herpes simplex virus after neurosurgery on thoracolumbosacral and lower lumbar nerve roots.

Patient 1: A 52-year-old man had a T-9 to L-1 laminectomy for microneurosurgical excision of an arteriovenous malformation.

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