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Cardiopulmonary Arrest: Pathophysiology and Neurologic Complications

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▸Requests for reprints should be addressed to Edward Bass, M.D.; 4710 North Habana, Suite 300; Tampa, FL 33614.

Tampa, Florida

©1985 American College of PhysiciansAmerican College of Physicians

Ann Intern Med. 1985;103(6_Part_1):920-927. doi:10.7326/0003-4819-103-6-920
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Cardiopulmonary arrest is a test of the brain's tolerance to global ischemia. New insights into the pathophysiology of global ischemia have led to the potential use of early prophylactic anticonvulsants, hypothermia, barbiturate coma, glucose manipulations, calcium-blocking agents, and hemodilution. A wide spectrum of neurologic sequelae may follow global ischemia, ranging from brain death, vegetative states, and impairment of higher intellectual function to syndromes of amnesia and cortical blindness, post-anoxic myoclonus, delayed leukoencephalopathy, and spinal stroke. The distinctive features of these sequelae and their pathophysiologic aspects are discussed. Special attention is given to brain death and prognostication.





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