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Hypomagnesemic Hypocalcemia Independent of Parathyroid Hormone

MARK L. GRABER, M.D.; and GERALD SCHULMAN, M.D.
[+] Article and Author Information

Grant support: in part by a Merit Review award of the Veterans Administration to Dr. Graber.

▸Requests for reprints should be addressed to Mark Graber, M.D.; SC 15-020, SUNY at Stony Brook; Stony Brook, NY 11794.


Veterans Administration Medical Center, Northport, New York; and State University of New York, Stony Brook, New York


Ann Intern Med. 1986;104(6):804-805. doi:10.7326/0003-4819-104-6-804
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This excerpt has been provided in the absence of an abstract.

Hypocalcemia, common in patients with magnesium depletion, has been attributed to either defective release of parathyroid hormone, resistance to the hormone's peripheral effects, or a combination of the two (1). We report a case that suggests that this hypocalcemia may simply represent a direct effect of magnesium depletion on bone independent of the effects of parathyroid hormone. A 68-year-old man presented with a thyroid mass. A biopsy specimen showed recurrence of a squamous cell carcinoma of a vocal cord that had been removed 4 years previously. His nutritional status was normal, as were all electrolyte determinations, including plasma concentrations of

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