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The Prevalence of Hepatitis C Virus Antibodies among Hemodialysis Patients

Jerome B. Zeldis, MD, PhD; Thomas A. Depner, MD; I. K. Kuramoto, BS; Robert G. Gish, MD; and Paul V. Holland, MD
[+] Article, Author, and Disclosure Information

Grant Support: Partial support by Dialysis Clinic, Inc., a nonprofit organization.

Requests for Reprints: Jerome B. Zeldis, MD, PhD, Division of Gastroenterology, 2110 Professional Building, 4301 X Street, University of California, Davis Medical Center, Sacramento, CA 95817.

Current Author Addresses: Dr. Zeldis: Gastrointestinal Division, Room 2110, 4301 X Street, Sacramento, CA 95817.

Dr. Depner: Renal Division, FOLBII, University of California, Davis Medical Center, Sacramento, CA 95817.

Mr. Kuramoto and Dr. Holland: Sacramento Medical Foundation Blood Center, 1625 Stockton Boulevard, Sacramento, CA 95816-7089.

Dr. Gish: Transplant Service, P.O. Box 7999/A-417, Pacific Presbyterian Medical Center, San Francisco, CA 94120.

Ann Intern Med. 1990;112(12):958-960. doi:10.7326/0003-4819-112-12-958
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This excerpt has been provided in the absence of an abstract.

The incidence of viral hepatitis in hemodialysis units has declined over the past 20 years with improved infection control strategies, including patient surveillance and segregation, plus improved prophylaxis with immune globulin, hepatitis B immune globulin, hepatitis B vaccines, and disinfectant procedures (1). Despite these measures, some hemodialysis patients have transient or prolonged elevations of their aminotransferase levels. Furthermore, end-stage liver disease is a major cause of death in long-term survivors of renal transplantation (2). Non-A, non-B hepatitis appears to be the major cause of hepatitis in hemodialysis units. Recently, a viral agent, hepatitis C virus (HCV), was isolated that may


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