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Multidrug-resistant Tuberculosis

Samuel W. Dooley, MD; William R. Jarvis, MD; William J. Marione, MD; and Dixie E. Snider Jr., MD, MPH
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Requests for Reprints: Samuel W. Dooley, MD, Centers for Disease Control, Mailstop E-10, 1600 Clifton Road NE, Atlanta, GA 30333.


Ann Intern Med. 1992;117(3):257-259. doi:10.7326/0003-4819-117-3-257
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This excerpt has been provided in the absence of an abstract.

As recently as 10 years ago, tuberculosis was rapidly disappearing from the United States. Reported cases of tuberculosis were decreasing an average of almost 6% each year, from over 84 000 cases in 1953 to 22 255 cases in 1984. Beginning in 1985, however, the decades-long decline in tuberculosis cases reversed dramatically, and from 1985 to 1991, an 18.4% increase was noted in reported cases (1) (Centers for Disease Control [CDC]. Unpublished data). One reason for this reversal is reactivation of latent tuberculous infection in persons infected with human immunodeficiency virus (HIV) (2, 3).

Against this background of the increasing

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