WALTER BORTZ, M.D.; EUGENE EISENBERG, M.D.; C. Y. BOWERS, M.D.; MANARD PONT, M.D.
Hypercalcemia in thyrotoxicosis was first reported largely as a medical curiosity.1 Since then additional cases have been reported. The finding that serum calcium returns to normal after treatment of thyrotoxicosis suggests a causal relationship. The true incidence of hypercalcemia with thyrotoxicosis is not known, but this combination occurs with sufficient frequency to warrant consideration of hyperthyroidism in the differential diagnosis of hypercalcemia. Because the per cent of renal tubular reabsorption of phosphate is affected by parathyroid function, its determination is useful in the evaluation of hypercalcemia.2 It is almost always low in hyperparathyroidism. In other causes of hypercalcemia—malignancy, vitamin D
BORTZ W, EISENBERG E, BOWERS CY, et al. DIFFERENTIATION BETWEEN THYROID AND PARATHYROID CAUSES OF HYPERCALCEMIA1. Ann Intern Med. 1961;54:610–619. doi: 10.7326/0003-4819-54-4-610
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Published: Ann Intern Med. 1961;54(4):610-619.
Endocrine and Metabolism, Fluid and Electrolyte Disorders, Nephrology.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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