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Original Research |1 June 1961

FAMILIAL RENAL TUBULAR ACIDOSIS1

RUSSELL E. RANDALL JR.; WILLIAM H. TARGGART, M.D.

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  • *Received for publication November 29, 1960.From the Medical Services, U. S. Air Force Hospital, Travis Air Force Base, Calif., the University of California Hospital, and the Department of Medicine, University of California, San Francisco, Calif.†Chief of Hospital Service, 1605th U. S. Air Force Hospital, APO 406, New York, N. Y.‡Research Fellow in Gastroenterology, University of Washington School of Medicine, Seattle 5, Wash.Requests for reprints should be addressed to Major Russell E. Randall, Jr., (MC) USAF, Chief, Hospital Service, 1605th U. S. Air Force Hospital, APO 406, New York, N. Y.
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Abstract

Acidosis of renal origin is an increased hydrogen ion concentration due to an impairment in the excretion of titratable acid and/or ammonium ion, to an excessive excretion of bicarbonate, or to a combination of these two defects.1-4 While all renal acidosis is presumably tubular in origin, the term "renal tubular acidosis" has come to represent (and will be so used in this text) that syndrome characterized by a hyperchloremic acidosis resulting from marked impairment in the ability to excrete an acid urine.3-15 The latter is significantly out of proportion to any impairment in glomerular filtration, and hence is not associated

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RANDALL RE, TARGGART WH. FAMILIAL RENAL TUBULAR ACIDOSIS1. Ann Intern Med. 1961;54:1108–1116. doi: 10.7326/0003-4819-54-6-1108

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Published: Ann Intern Med. 1961;54(6):1108-1116.

DOI: 10.7326/0003-4819-54-6-1108

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