DOROTHY GILL, M.D.
This content is PDF only. Please click on the PDF icon to access.
Hypercalcemia, hypophosphatemia, and increased excretion of calcium and phosphorus in the urine characterize the metabolic changes in clinical hyperparathyroidism. The appearance of a high blood calcium in such conditions as multiple myeloma and metastatic carcinoma of bone has led to speculation as to whether this is simply incident to diseases causing rapid bone destruction or whether true hyperactivity of the parathyroid glands exists. Bulger and Barr1 were inclined toward the latter theory, as it is known (a) that it is difficult to raise the serum calcium even by giving large quantities of calcium by mouth, and (b) that the parathyroids
GILL D. MULTIPLE MYELOMA SIMULATING HYPERPARATHYROIDISM*. Ann Intern Med. 1946;24:1087–1093. doi: https://doi.org/10.7326/0003-4819-24-6-1087
Download citation file:
Published: Ann Intern Med. 1946;24(6):1087-1093.
Endocrine and Metabolism, Hematology/Oncology, Multiple Myeloma, Parathyroid Disorders.
Results provided by:
Copyright © 2020 American College of Physicians. All Rights Reserved.
Print ISSN: 0003-4819 | Online ISSN: 1539-3704
Conditions of Use