WILLIAM BLOOM, M.D.
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The pathogenesis of chronic hypertrophic osteoarthropathy remains unsolved. The clinical entity is obscured rather than clarified by a multiplicity of unsatisfactory hypotheses.1
Within the last five or six years evidence has appeared in the literature that would seem to point to a more logical understanding of this uncommon disease. That the proper approach to this subject is an endocrine one may be suspected by emphasizing the common characteristics of three syndromes which may be confused clinically, viz.—acromegaly, pachyperiostosis of Touraine and Gallé2 and hypertrophic pulmonary osteoarthropathy. The first condition is of definite endocrine origin. The second is very often considered
BLOOM W. PITUITARY IMPLICATIONS IN HYPERTROPHIC PULMONARY OSTEOARTHROPATHY1. Ann Intern Med. ;29:361–370. doi: 10.7326/0003-4819-29-2-361
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Published: Ann Intern Med. 1948;29(2):361-370.
Endocrine and Metabolism, Neurology, Pituitary Disorders.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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