PETER GABERMAN, M.D.; DONALD H. ATLAS, M.D., F.A.C.P.; ERWIN M. KAMMERLING, M.D.; LEE EHRLICH, M.D.; JULIEN ISAACS, M.D.
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Azotemia from nonrenal disease states has received much attention in the literature in recent years. Such azotemia has been described in a multitude of conditions— medical, surgical, obstetrical, physical and allergic. A variety of names has been given to this syndrome of renal insufficiency, among them acute Bright's disease, acute tubular nephritis, prerenal azotemia, lower nephron syndrome, traumatic kidney, shock kidney and crush syndrome. Since the pathogenesis and the pathologic lesions are equivocal and the only discernible common factor seems to be renal anoxia, we consider the best description to be renal anoxia syndrome, as suggested by Maegraith et al.1
GABERMAN P, ATLAS DH, KAMMERLING EM, et al. RENAL ANOXIA SYNDROME: A REVIEW AND REPORT OF 22 CASES*. Ann Intern Med. 1951;35:148–168. doi: https://doi.org/10.7326/0003-4819-35-1-148
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Published: Ann Intern Med. 1951;35(1):148-168.
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