H. J. L. M.
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Vasospasm has become a clinical cliché—a glib device to explain what at first seems to be otherwise inexplicable. Time and again we encounter, in books, in papers and in consultation, phrases that echo Osler's words, "the ephemeral nature of the attacks can scarcely be explained in any other way." Arterial spasm has become entrenched in our minds and texts as the underlying mechanism of transient cerebral syndromes. That this acceptance is hasty, uncritical and unnecessary, becomes evident from a consideration of known facts.
Cogent arguments against the acceptance of angiospasm as the mechanism of transient hypertensive crises have been advanced
M. HJL. THE QUESTION OF CEREBRAL ANGIOSPASM. Ann Intern Med. 1952;36:1129–1135. doi: https://doi.org/10.7326/0003-4819-36-4-1129
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Published: Ann Intern Med. 1952;36(4):1129-1135.
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