MATTHEW TAUBENHAUS, M.D., F.A.C.P.; ORMAND C. JULIAN, M.D., F.A.C.S.; JAKUB SCHLICHTER, M.D.; MARGARET LITTMAN, M.D.
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Interest in the problem of portal hypertension has been renewed since successful new operative procedures have been devised to relieve the increased pressure in the portal system.1, 2, 3, 4, 5 The recognition of this syndrome presents no major problem, particularly if a stage is reached in which pronounced signs and symptoms are present. These consist of a history of hematemesis and/or melena, the presence of ascites, splenomegaly and enlarged collateral veins in the abdominal wall, and x-ray evidence of esophageal varices. The anatomic basis for this condition has been described6 the experimental work on this subject reviewed,7, 8 and
TAUBENHAUS M, JULIAN OC, SCHLICHTER J, et al. UNUSUAL ETIOLOGIES OF PORTAL SYSTEM HYPERTENSION (TORULOSIS, PHLEBOSCLEROSIS AND RETROPERITONEAL CHRONIC INFLAMMATION)*. Ann Intern Med. 1954;40:313–326. doi: https://doi.org/10.7326/0003-4819-40-2-313
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Published: Ann Intern Med. 1954;40(2):313-326.
Cardiology, Coronary Risk Factors, Hypertension, Infectious Disease, Nephrology.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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