S. K. FINEBERG, M.D., F.A.C.P.; A. ALTSCHUL, M.D., F.A.C.P.
Gout may be succinctly defined as a disorder of purine metabolism characterized by hyperuricemia and a typical form of recurrent acute arthritis which often eventually is associated with urate deposits in cartilages, bone, joint spaces, bursae, tendons, kidneys and other tissues.1 Although the existence of a "gouty nephritis" as a "complication" of this disease has long been recognized,2, 3, 4, 5 the high incidence of renal damage and its true place as an integral part of the whole disease process do not appear to be generally appreciated. Careful study, however, reveals that the metabolic error in gout produces not only
FINEBERG SK, ALTSCHUL A. THE NEPHROPATHY OF GOUT1. Ann Intern Med. 1956;44:1182–1194. doi: 10.7326/0003-4819-44-6-1182
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Published: Ann Intern Med. 1956;44(6):1182-1194.
Gout, Nephrology, Rheumatology.
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