GIUSEPPE FOLLI, M.D.; VICTOR E. POLLAK, M.B., M.R.C.P.E.; Ross T. W. REID, M.D.; CONRAD L. PIRANI, M.D.; ROBERT M. KARK, F.A.C.P., F.R.C.P.
The nephrotic syndrome is associated with a number of disease processes, which produce a variety of pathologic lesions in the kidney. The histology of many of these disease processes, including amyloidosis, lupus nephritis, proliferative glomerulonephritis with epithelial crescents, and renal vein thrombosis, is well known and widely accepted. On the other hand, the histologic distinction between lipoid nephrosis and membranous glomerulonephritis is still the subject of controversy. Few now agree with Munk's original description of the pathology of lipoid nephrosis,1 namely, fatty changes in the tubules and—at least by light microscopy—normal glomeruli. Many agree with Allen2 that ". . .
FOLLI G, POLLAK VE, REID RTW, et al. ELECTRONMICROSCOPIC STUDIES OF REVERSIBLE GLOMERULAR LESIONS IN THE ADULT NEPHROTIC SYNDROME*. Ann Intern Med. 1958;49:775–795. doi: https://doi.org/10.7326/0003-4819-49-4-775
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Published: Ann Intern Med. 1958;49(4):775-795.
Nephrology, Nephrotic Syndrome.
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