D. P. STABLES, M.B., B. CHIR.; N. W. LEVIN, M.B., F.C.P. (S.A.); A. H. RUBENSTEIN, M.B., B. CH.; M. E. BERK, M.B., F.F.R., D.M.R.D.; C. ABRAHAMS, M.B., F.F. PATH. (S.A.)
The concept of renal osteodystrophy, originally introduced by Liu and Chu (1), is now widely recognized, and the main features have been well described (2-4). Recalcification therapy with vitamin D has been observed to produce both symptomatic relief and radiological healing both of rickets and, in the majority of cases, of osteitis fibrosa, if the treatment is sufficiently prolonged (3, 5). Where, however, the changes of secondary hyperparathyroidism predominate and metastatic calcification is present, administration of vitamin D involves the risk of a further rise in the already high serum calcium-phosphorus product and, consequently, of a dangerous increase in metastatic
STABLES DP, LEVIN NW, RUBENSTEIN AH, et al. Parathyroidectomy for Hypercalcemic Crisis in Renal Osteodystrophy. Ann Intern Med. 1964;61:531–539. doi: 10.7326/0003-4819-61-3-531
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Published: Ann Intern Med. 1964;61(3):531-539.
Endocrine and Metabolism, Fluid and Electrolyte Disorders, Nephrology, Parathyroid Disorders.
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