DONNA K. MCCURDY, M.D.; GIBBONS G. CORNWELL III, M.D.; VICTOR J. DEPRATTI, M.D.
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Renal tubular acidosis is a syndrome in which a specific inability of the renal tubule to lower urine pH normally is associated with renal bicarbonate wastage and, ultimately, hyperchloremic acidosis. Other common but by no means invariable manifestations of the syndrome include hypokalemia due to renal potassium wastage, nephrolithiasis and nephrocalcinosis, a vasopressin-resistant nephrogenic diabetes insipidus, hypercalciuria, decreased citrate excretion, and osteomalacia (1-4). Although the primary form of this syndrome occurs both familially and sporadically, unassociated with other diseases, secondary renal tubular acidosis may develop in the course of a number of disease states that in various ways may lead
MCCURDY DK, CORNWELL GG, DEPRATTI VJ. Hyperglobulinemic Renal Tubular Acidosis: Report of Two Cases. Ann Intern Med. 1967;67:110–117. doi: https://doi.org/10.7326/0003-4819-67-1-110
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Published: Ann Intern Med. 1967;67(1):110-117.
Hematology/Oncology, Hospital Medicine, Nephrology.
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Print ISSN: 0003-4819 | Online ISSN: 1539-3704
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