J.R. TAGG, PH.D.; G. PETERS, M.D.; E.D. GRAY, PH.D.; L.W. WANNAMAKER, M.D.
This content is PDF only. Please click on the PDF icon to access.
To the editor: Although an etiologic association between toxic shock syndrome and vaginal colonization with Staphylococcus aureus is generally accepted as being beyond doubt, many questions regarding the pathogenesis of the syndrome remain unanswered. The ability of potentially pathogenic bacteria to colonize body surfaces may be enhanced by their production of inhibitory substances that restrict the growth of other bacteria (1). Evaluation of bacteriocin production by strains of S. aureus associated with toxic shock syndrome has been our primary interest. These proteinaceous antibiotic agents have inhibitory activity directed predominantly against other bacteria of either the same or closely-related species (2).
TAGG J, PETERS G, GRAY E, et al. Bacterial Interference and Toxic Shock Syndrome. Ann Intern Med. 1983;99:879–880. doi: 10.7326/0003-4819-99-6-879_2
Download citation file:
Published: Ann Intern Med. 1983;99(6):879-880.
Copyright © 2019 American College of Physicians. All Rights Reserved.
Print ISSN: 0003-4819 | Online ISSN: 1539-3704
Conditions of Use