NANCY S. NOWLIN, M.D.; JAMES E. BRICK, M.D.; DANA J. WEAVER, M.D.; DEBORAH A. WILSON, M.D.; HOWARD L. JUDD, M.D.; JOHN K. H. LU, Ph.D.; HAROLD E. CARLSON, M.D.
Hormonal, neurologic, and vascular factors affecting potency were evaluated in 10 men with scleroderma and in 10 age-matched men with rheumatoid arthritis. Impotence was reported by 6 of the patients with scleroderma and none with rheumatoid arthritis. Studies of serum testosterone, free testosterone index, follicle-stimulating hormone, luteinizing hormone, prolactin, estradiol, thyroxine, and thyrotropin did not show a hormonal basis for impotence in any patient. Neurologic causes were not found on physical examination. Penile blood pressures were markedly abnormal in 4 impotent patients, intermediate in 2 impotent and 3 potent patients, and normal in 11 potent patients. A history of claudication and diminished ankle blood pressures indicated large vessel disease in 2 impotent patients; the remaining 4 impotent men had normal ankle pressures, suggesting that their poor penile blood pressures and impotence were due to small vessel disease, perhaps the small artery lesions of scleroderma.
NOWLIN NS, BRICK JE, WEAVER DJ, et al. Impotence in Scleroderma. Ann Intern Med. 1986;104:794–798. doi: https://doi.org/10.7326/0003-4819-104-6-794
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Published: Ann Intern Med. 1986;104(6):794-798.
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